This webpage is based on the information obtained from recent medical and scientific advances, for example:
* Monosodium urate (MSU) crystals, a compound of sodium and uric acid, which cause gout attack can build up in the joints years or even decades before the first gout attack. They are coated by certain proteins and are imbedded innocently in joints and other tissues.
* Acute gout attacks occur when the coatings of the MSU crystals are shedded to expose the raw crystals by certain triggers. When the crystals are recoated, gout subsides.
* Sudden increase or decrease of the blood uric acid level can cause the significant in-flow or out-flow of MSU between joint fluids and the blood, and cause the shedding of the coatings to trigger gout attacks. There are other triggers.
* Uric acid is more soluble in alkaline fluids; MSU in acidic fluids. Therefore low pH in the body can make uric acid less soluble, more difficult to excrete, and cause gout and uric acid stones in the kidneys and urinary tracks.
* Most of the patients who took Allopurinol to lower the uric acid level to prevent gout were found to have MSU crystals still in their joints months or years later.
* High level of uric acid in the blood (hyperuricemia) is neither the necessary nor the sufficient condition for gout. About 80~92% of the hyperuricemic people never have gout in their life time.
* During the gout attack, about 10%~50% of the hyperuricemic gout patients have normal or low blood uric acid levels because more uric acid is excreted during the attack. Therefore, the diagnosis based on the blood uric acid tests done during the gout attacks can be erroneous. Joint fluid tests can identify the MSU crystals and other substances and organisms to diagnose gout and/or other diseases.
* The strict low purine diet can reduce the blood uric acid level by only about 1 mg/dL (normal < 7mg/dL) which is quite small as compared with what can be achieved by the use of uric acid lowering drugs -- 2.5~3.5 mg/dL reduction. About 50%~70% of the patients who normalized their blood uric acid level with drugs were found to have MSU crystals still in their joints. Therefore it is more important to avoid the gout triggers than merely to be on low purine diet.
* I accidentally cured my 27-year old worsening gout by drinking baking soda in water while taking Indomethacin and Benemid during a gout attack. I have been free of gout for about 4.5 years (in January 2007) without taking any gout medicine and have been on unrestricted diet.
Tuolta pistää silmään heti nuo kohdat joissa sanotaan, että virtsahapon määrällä ei ole suurtakaan merkitystä kihdin kanssa ja toisaalta kihdin hoitoon ruokavalio ei ole kovinkaan tehokas tapa. Omat kokemukseni puoltavat tällä sivulla esitettyjä päätelmiä.
Ruokasoodaa en ole kokeillut. Ei ole ollut kohtauksiakaan. Villasukat ovat edelleen kihdin hoitoon yksi parhaista lääkkeistä.
Sivustolla oli myös tutkimusviite, jota olisi hyvä tutkia hieman enemmän.
3.30 Icing is beneficial and heating is not, in treating gout attacks and may help diagnose gou.(2/25/07) The recommendations on whether icing or heating is beneficial to treat gout attack have been contradictory in the gout literature. Not any more. The data in a recent study has shown that significantly higher percentage of gout patients have found icing the gout attack sites helped relieve their pain as compared with patients with rheumatoid and other forms of arthritis. None of the gout patients benefited from heating the affected joints during the attacks. The result is so significant that the report goes so far as to suggest using icing to diagnose gout during the attacks, i.e., if one feels better by icing the attack site, it's gout; if not, it's not gout and is some other forms of arthritis. For more info see the original report at:http://www.jclinrheum.com/
Jos kylmä auttaa, niin se on kihti ja jos lämmin auttaa, niin se on joku muu tulehdus. Onko minulla siis joku muu nivelsairaus, joka on tulkittu liian hätäisesti kihdiksi, koska kylmä pahentaa ja lämmin auttaa.
Jos tältä listalta 6 täsmää 13:sta, niin se on todennäköisesti kihti.
1. More than one attack of acute arthritis (joint inflammation).
2. Maximum inflammation developed within 1 day.
3. Attack of a single joint.
4. Redness over joints.
5. Metatarsophalangeal joint (the joint between the second and third bones) of the big toe painful or swollen.
6. Attack of the the big toe in one foot.
7. Attack of heel joint in one foot.
8. Tophus, deposits of MSU crystals in tissues (proven or suspected).
10. Asymmetric swelling within a joint on x ray.
11. Subcortical cysts without erosions on x ray.
12. MSU crystals in joint fluid during attack.
13. Joint fluid culture negative for organisms during attack.
Kohdat 1, 2, 3, 4, 5, 6, 8 ja 9 osuu ja on tutkittu. Röntgeniin on lähete. Jostain syystä lääkärit eivät, ainakaan täällä maalla, tutki nivelnestettä. Sehän olisi kaiketi varmin tapa selvittää onko kristalleja vai ei.